Yung6ix was involved in an accident around 4am today. According to reports, he’s fine, please disregard any rumour stating otherwise although his Ranger Rover Jeep is Badly Damaged.
Every year, almost 200,000 women in America are diagnosed with breast cancer, making it the most common cancer diagnosed in women aside from skin cancer. Approximately one in every eight females will develop breast cancer at some point in her life. Only lung cancer claims more lives than breast cancer in women overall, but breast cancer serves as the leading cause of cancer-related deaths among some populations, such as Hispanic women.
There are significant differences between breast cancers that occur before and after menopause. Hereditary factors figure prominently in premenopausal breast cancers, while most breast cancers that develop after menopause have far less family history involvement.
Without an inherited predisposition, many women will look at their health behaviors and lifestyle choices as suggestive of their risks. Drinking alcohol, smoking, eating poorly or being obese, and a lack of exercise may contribute to breast cancer occurrence. Some studies have implicated alcohol consumption and obesity for increasing risk; another found that regular exercise can lower the incidence of breast cancer.
Still, scientists have yet to explain how some women with virtually identical risk factors can have such different outcomes, or why women without any risk factors end up developing breast cancer.
Breast cancer mortality rates have declined in recent years thanks primarily to increased awareness and early detection. Medicines continue to improve and treatment options are expanding, but women still need to be vigilant in order to increase their chances of success should they develop breast cancer.
Types of Breast Cancer
There are a number of different cancers that can occur in the breast, and treatment and prognosis vary according to the type. Generally, breast cancers are grouped according to where they first develop.
These types of cancer first develop in the milk-producing ducts of the breast. These types of breast cancer are considered the earliest forms of breast cancer and the most common kinds, and they can be either invasive (invasive ductal carcinoma) or noninvasive (ductal carcinoma in situ, or DCIS). “Invasive” means that the cancer has spread from the original site to nearby breast tissue and/or lymph nodes and other parts of the body.
These cancers develop in the cells lining the lobules that produce milk, are the second most common type of breast cancer. These are always considered invasive because they spread to surrounding tissue. (Lobular carcinoma in situ is sometimes referred to as cancer but actually stays confined to the lobules or milk glands. However, this type serves as an indicator that you have a higher chance of developing breast cancer in the future.)
Other breast cancers
Other, rarer types of cancer include:
Breast sarcomas are cancers that start from connective tissues of the breast, such as muscle, fat, or blood vessels.
Inflammatory breast cancer blocks the lymphatic vessels in the breast, causing severe inflammation.
Paget’s Disease of the breast is a rare form of breast cancer where cancer cells gather in or around the nipple.
Male breast cancer is very rare, but can occur. When it does, it is usually ductal carcinoma.
Breast Cancer Causes & Risk Factors
Medical scientists do not fully understand what causes breast cancer. Like other cancers, it is marked by a rapid production of some cells that amass into a tumor.
Scientists have isolated some genes that, if mutated, seem to cause breast cancer: breast cancer gene 1 (BRCA1) and breast cancer gene 2 (BRCA2). These genes comprise up to 10 percent of breast cancer patients. However, experts don’t know what makes up the biochemical cascade that causes changes in a woman’s genes.
Here are some other risk factors that may lead to breast cancer:
Unavoidable Risk Factors
Women acquire breast cancer 100 times more often than men because of their hormones, specifically estrogen and progesterone. The exposure of breast cells in a woman to these growth-drivers accounts for the huge difference.
More than two-thirds of invasive breast cancers are found in women age 55 and older. Only one-eighth is found in women under age 45. Male breast cancer most often strikes those between age 60 and 70.
Women have a higher risk of breast cancer if they have a first-degree blood relative (such as a mother, sister or daughter) who has been diagnosed with the disease. Two first-degree relatives increase a woman’s risk by five times.
The genetic mutations of BRCA1 and BRCA2 genes are, by far, the most prevalent causes of inherited breast cancer. They account for almost the entire 10 percent of hereditary cases. BRCA mutations may spike the risk to 80 percent in some families. In the U.S., women of Eastern European Jewish background have been found to have disproportionately high BRCA mutations. Other genes that are implicated in hereditary breast cancer, though rare, include ATM, p53, CHEK2, PTEN, and CDH1.
Women of European background are most likely to be diagnosed with breast cancer. However, African American women are more likely to die from the disease. Breast cancer is also the No. 1 cause of cancer death in Hispanic women.
Some physical characteristics have shown to increase a woman’s risk for breast cancer. These include:
Periods that begin before age 12
Going through menopause after 55 (also called “delayed menopause”)
Having dense breasts
Obesity or being overweight
High alcohol consumption
Not having children
Having your first child after 35
Not breastfeeding (according to some studies)
Taking birth control pills
Using Hormone Replacement Therapy
Risk Factors For Men
High levels of estrogen (due to cirrhosis of the liver, for example) or estrogen-related drugs (such as for prostate cancer)
Exposure to radiation
Over age 60
High alcohol consumption
Inherited gene mutations
Family history of breast cancer
TOKYO: Japanese scientists say they have found
a way to slow down the ageing process in flowers
by up to a half, meaning bouquets could remain
fresh for much longer.
Researchers at the National Agriculture and Food
Research Organisation in Tsukuba, east of Tokyo,
said they had found the gene believed to be
responsible for the short shelf-life of flowers in
one Japanese variety of morning glory.
“Morning glory” is the popular name for a
hundreds of species of flowering plants whose
short-lived blooms usually unfold early in the day
and are gone by nightfall.
By suppressing the gene — named
“EPHEMERAL1” — the lifespan of each flower was
almost doubled, said Kenichi Shibuya, one of the
lead researchers in a study carried out jointly with
Kagoshima University in southern Japan.
“Unmodified flowers started withering 13 hours
after they opened, but flowers that had been
genetically modified stayed open for 24 hours,” he
This means the plant has fresh purple flowers
alongside the paler blooms from the previous day,
“We have concluded that the gene is linked to
petal ageing,” Shibuya told AFP by telephone on
The finding could lead to developing methods to
extend the life of cut flowers, he added.
“It would be unrealistic to modify genes of all
kinds of flowers but we can look for other ways to
suppress the (target) gene… such as making cut
flowers absorb a solution that prevents the gene
from becoming active,” Shibuya said.
For some flowers, such as carnations, florists
currently use chemicals to inhibit ethylene, a
plant hormone which sometimes causes blooms
But ethylene is not involved in the ageing of some
popular flowers, such as lilies, tulips and irises.
A gene similar to EPHEMERAL1 could be
responsible for petal ageing in these plants,
Shibuya said, meaning the ability to suppress it
would extend their life.
Our modern technology touches the lives and
connects them together no matter who they are
and where they belong. But when it comes to the
communication based on how effectively you use
either it may construct or destruct the
Emoticons are powerful means of communication
that most of us madly rely on today and I guess
this is why you have arrived here with the query
what does -p mean in a text message. Without
the inclusion of emoticons language loses a lot.
Communicating through plain text is one of the
poorest means of communication. It bores the
recipients. Never do that when you converse with
your friends and family. As adding emoticon and
inflection brings an extra meaning to the text
communication, people find it as more intimate.
When Exactly These Emoticons Were Invented?
Can you guess the very first emoticon which was
created 35 years ago? Yes you are right. It’s the
smile emoticon. Anyhow, the original “yellow
smiley” was created by an American graphic
designer Harvey Ross Ball in 1963 for “the State
Mutual Life Assurance Company” which is now
known with the name Hanover Insurance.
There Is a Story Behind Emoticons
After the acquisition of Guarantee Mutual
Company due to the difficult merges morale
among the employees gone low. So to spread the
smile and spirit among the employees, the
insurance company commissioned ball to create a
graphic design. He finished the design in not
more than 10 minutes and he was paid with $45
for his work. When he created the smiley he never
knew that it was going to be his master piece.
Since then smiley emoticon has become very
popular and has gained the recognition of
International smile icon.
So now let’s get back to your search what does –
P mean in a text message?
: P is the same as :-P.
It resembles a funny face
with a tongue sticking out of its mouth in
It can be pictured as below
-P or 😛
Today’s technology (from mobile apps to social
media websites) has made emoticons to become
an inseparable part of our text communication.
Some of the most commonly used emoticons are
: ) or
Very happy (Double chin)
Really Big Grin
Surprised or Shocked
Perplexed or Confused
Big Eyes Smile
i hope now you have got the idea on the meaning
of -p in texting. Explore more emoticons to your
text communications and make it fun filled and
Cervical cancer is a type of cancer that occurs in
the cells of the cervix — the lower part of the
uterus that connects to the vagina. Various
strains of the human papillomavirus (HPV), a
sexually transmitted infection, play a role in
causing most cases of cervical cancer.
When exposed to HPV, a woman’s immune
system typically prevents the virus from doing
harm. In a small group of women, however, the
virus survives for years, contributing to the
process that causes some cells on the surface of
the cervix to become cancer cells.
You can reduce your risk of cervical cancer by
undergoing screening tests and receiving a
vaccine that protects against HPV infection.
Most cervical cancer is caused by a virus called
human papillomavirus , or HPV. You can get HPV
by having sexual contact with someone who has
it. There are many types of the HPV virus. Not all
types of HPV cause cervical cancer. Some of
them cause genital warts , but other types may
not cause any symptoms.
Most adults have been infected with HPV at some
time. An infection may go away on its own. But
sometimes it can cause genital warts or lead to
cervical cancer. That’s why it’s important for
women to have regular Pap tests. A Pap test can
find changes in cervical cells before they turn into
cancer. If you treat these cell changes, you may
prevent cervical cancer.
Abnormal cervical cell changes rarely cause
symptoms. But you may have symptoms if those
cell changes grow into cervical cancer. Symptoms
of cervical cancer may include:
Bleeding from the vagina that is not normal,
such as bleeding between menstrual periods,
after sex, or after menopause.
Pain in the lower belly or pelvis.
Pain during sex.
Vaginal discharge that isn’t normal.
As part of your regular pelvic exam, you should
have a Pap test. During a Pap test, the doctor
scrapes a small sample of cells from the surface
of the cervix to look for cell changes. If a Pap
test shows abnormal cell changes, your doctor
may do other tests to look for precancerous or
cancer cells on your cervix.
Your doctor may also do a Pap test and take a
sample of tissue ( biopsy ) if you have symptoms
of cervical cancer, such as bleeding after sex.
The treatment for most stages of cervical cancer
Surgery, such as a hysterectomy and removal
of pelvic lymph nodes with or without removal
of both ovaries and fallopian tubes.
If you are age 26 or younger, you can get the HPV
vaccine, which protects against two types of HPV
that cause most cases of cervical cancer.
The virus that causes cervical cancer is spread
through sexual contact. The best way to avoid
getting a sexually transmitted infection is to not
have sex. If you do have sex, practice safer sex,
such as using condoms and limiting the number
of sex partners you have.
smoking – women who smoke are twice as
likely to develop cervical cancer than women
who don’t; this may be caused by the harmful
effects of chemicals found in tobacco on the
cells of the cervix
having a weakened immune system – this can
be the result of taking certain medications,
such as immunosuppressants, which are used
to stop the body rejecting donated organs, or
as a result of a condition such as HIV/AIDS
taking the oral contraceptive pill for more than
five years – women who do this are thought to
have twice the risk of developing cervical
cancer than those who do not take the pill,
although it is not clear why this is
having children (the more children you have,
the greater your risk) – women who have two
children have twice the risk of getting cervical
cancer compared with women who do not have
The reason for the link between cervical cancer
and childbirth is unclear. One theory is that the
hormonal changes that occur during pregnancy
could make the cervix more vulnerable to the
effects of HPV.
President-General of the Nigeria Football Supporters Club, Rafiu Ladipo, has publicly criticized the members of the Nigeria squad that were dumped out of the 2014 World by France.
Paul Pogba struck in the 79th minute, while Joseph Yobo also scored an own goal, as the African champions failed to progress past the round of 16 again.
Ladipo admitted that he was full of optimism ahead of the tournament, but was disappointed in the manner which he Eagles crashed out.
“I was one of those who came here confident that we will surpass our previous World Cup record. Crashing out in the second round was to me not good enough,” Ladipo said.
“We were there before 16 years ago and attaining that same feat here is really no big deal. Keshi and his boys had the opportunity to make history but they blew it.”
“The match against France was winnable if only the team had remained focused before the game. But rather than look at the bigger picture and putting national interest first, the boys were more interested in money, which they got before the game.
“And with money in their pocket they felt whatever result they get in the game against France won’t be a big deal.”
Ladipo also took a swipe at Stephen Keshi, stating that the Big Boss did not pick the right squad for the competition.
“Keshi did not help himself by coming to Brazil with a good number of players who had no business being in the Eagles squad and leaving out players like Ike Uche and Haruna Lukman,” he concluded.
Britain is to lead a global effort to combat
antibiotic-resistant superbugs that threaten to
knock medicine “back into the dark ages,” Prime
Minister David Cameron said Wednesday.
LONDON: Britain is to lead a global effort to
combat antibiotic-resistant superbugs that
threaten to knock medicine “back into the dark
ages,” Prime Minister David Cameron said
Unless new antibiotics are found, the rise of
untreatable bacteria threatens a “unthinkable
scenario” where once-treatable minor infections
become fatal, he said.
As bacteria and viruses develop resistance to
existing antibiotics, the effectiveness of such
treatments naturally diminishes.
Cameron privately raised the issue with US
President Barack Obama and German Chancellor
Angela Merkel at the G7 summit last month and
believes he has their agreement for a co-
ordinated international push.
“This is not some distant threat but something
happening right now,” Cameron told The Times
“If we fail we are looking at an almost
unthinkable scenario where antibiotics no longer
work and we are cast back into the dark ages of
medicine where treatable infections and injuries
will kill once again.
“That simply cannot be allowed to happen and I
want to see a stronger, more coherent global
Jim O’Neill, the former chief economist of US
investment bank Goldman Sachs, is to lead an
international expert group aiming to spark the
development of a new generation of antibiotics.
He has been tasked with considering how
developing countries can be encouraged to
improve control of existing antibiotics, of which
only five new types have been introduced since
Scientific advisers “are all saying this is one of
the most serious health problems the world
faces,” Cameron said.
“For many of us we only know a world where
infections or sicknesses can be quickly remedied
by a visit to the doctor and a course of
“But that protection is at risk as never before.
Resistance to antibiotics is now a very real and
The initial £500,000 (US$860,000, 630,000 euros)
cost of the work will be met by the Wellcome
Trust biomedical research charity.
Drug-resistant bacteria, viruses and parasites
threaten not only the ability to treat deadly
infections but also most aspects of modern
medicine, said the trust’s director Jeremy Farrar.
“From cancer treatment to Caesarean sections,
therapies that save thousands of lives every day
rely on antibiotics that could soon be lost,” he
“We are failing to contain the rise of resistance,
and failing to develop new drugs to replace those
that no longer work. We are heading for a post-
Chief medical officer Sally Davies, the British
government’s principal medical adviser, said that
across Europe, at least 25,000 people a year die
from infections which are resistent to antibiotics.
She said adults had two kilogrammes of bacteria
in their bodies, “but they can infect us if our
immune systems are not so good”.